2018PubMedOpen access

[Establishment and optimization of hepatocyte steatosis model].

Junhong Gao, X. J. Li, W. Zhang, Y. S. Li, Hao Tang

Open full text 0 citations

Abstract

L02 cells were successfully exposed to high fat and ethanol, and the hepatocyte steatosis model was established and optimized, suggesting that the occurrence of hepatic cell steatosis was related to the up-regulation of PPAR-γ and AP-2.

About this research paper

What this paper is about

L02 cells were successfully exposed to high fat and ethanol, and the hepatocyte steatosis model was established and optimized, suggesting that the occurrence of hepatic cell steatosis was related to the up-regulation of PPAR-γ and AP-2.

Why it matters

A significance statement is not available in the OpenAlex record.

Key contribution

A contribution statement is not available in the OpenAlex record.

Method / approach

Method details are not available in the OpenAlex metadata.

Main findings

Findings are not separately available in the OpenAlex metadata.

Limitations

Limitations are not available in the OpenAlex metadata.

Applications

Application details are not available in the OpenAlex metadata.

Available abstract

L02 cells were successfully exposed to high fat and ethanol, and the hepatocyte steatosis model was established and optimized, suggesting that the occurrence of hepatic cell steatosis was related to the up-regulation of PPAR-γ and AP-2.

Key concepts: Steatosis, Oleic acid, Oil Red O, Hepatocyte, Lipid droplet, Fetal bovine serum, Ethanol, Chemistry

Related papers

Back to paper searchBrowse research topicsOriginal source
[Establishment and optimization of hepatocyte steatosis model]. — Research Paper | ScholarLens