2002•Chinese Journal of Thrombosis and HemostasisRequires access

Research Between Triglyceride and Plasminogen Activity on Abnormal in Patients with Cerebral Infarction

Wajvg Mei-lian

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Abstract

Objective: To study the risk factor of triglyceride and plasminogen activity on abnormal in patients with cerebral infarction. Methods: Using ELISA and enzyme methods, the content of tissue plasminogen activitor(t-PA) plasminogen activitorinhibator-1(PAI-1) and triglyceride(TG) in 51 patients with cerebral infarction, and 50 normal controls were analyse. Results: The changes of high TG, PAI-1 and low level t-PA were most significant abnormal charctor of cerebral infarction. Conclusion: High TG may an important role in the pathogenesis of cerebral infarction by in duing fibrinolytic dysufunction.

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Objective: To study the risk factor of triglyceride and plasminogen activity on abnormal in patients with cerebral infarction. Methods: Using ELISA and enzyme methods, the content of tissue plasminogen activitor(t-PA) plasminogen activitorinhibator-1(PAI-1) and triglyceride(TG) in 51 patients with cerebral infarction, and 50 normal controls were analyse. Results: The changes of high TG, PAI-1 and low level t-PA were most significant abnormal charctor of cerebral infarction. Conclusion: High TG may an important role in the pathogenesis of cerebral infarction by in duing fibrinolytic dysufunction.

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Available abstract

Objective: To study the risk factor of triglyceride and plasminogen activity on abnormal in patients with cerebral infarction. Methods: Using ELISA and enzyme methods, the content of tissue plasminogen activitor(t-PA) plasminogen activitorinhibator-1(PAI-1) and triglyceride(TG) in 51 patients with cerebral infarction, and 50 normal controls were analyse. Results: The changes of high TG, PAI-1 and low level t-PA were most significant abnormal charctor of cerebral infarction. Conclusion: High TG may an important role in the pathogenesis of cerebral infarction by in duing fibrinolytic dysufunction.

Key concepts: Medicine, Triglyceride, Cerebral infarction, Internal medicine, Pathogenesis, Infarction, Cardiology, Endocrinology

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