Quantitative study of the correlation between acute coronary syndromes and cardiac troponin I
Xing Qichong
Abstract
Xing Qichong
Abstract
Objective: To find out a quantitative correlation, if any, between the severity of coronary artery disease, assessment of left ventricular function and peak cTnI. Methods: Fifty-one patients underwent acute coronary syndrome(ACS) were involved. According to the severity of coronary artery disease, we divided them into three groups. Peak cTnI level and ultrasound cardiogramwere needed in every patient. Major cardiovascular events were observed for a month. Results: The peak cTnI levels of the three groups were statistically different. The peak cTnI levels whose LVEF40% were statistically higher than those whose LVEF≥40%. The peak cTnI levels with MCE were statistically higher than those without MCE. Conclusion: There was significant relationship between CAG results, LVEF levels and peak cTnI levels. Patients who had MCE had higher cTnI level. Thus, peak cTnI had diagnostic and prognostic value for ACS.
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Objective: To find out a quantitative correlation, if any, between the severity of coronary artery disease, assessment of left ventricular function and peak cTnI. Methods: Fifty-one patients underwent acute coronary syndrome(ACS) were involved. According to the severity of coronary artery disease, we divided them into three groups. Peak cTnI level and ultrasound cardiogramwere needed in every patient. Major cardiovascular events were observed for a month. Results: The peak cTnI levels of the three groups were statistically different. The peak cTnI levels whose LVEF40% were statistically higher than those whose LVEF≥40%. The peak cTnI levels with MCE were statistically higher than those without MCE. Conclusion: There was significant relationship between CAG results, LVEF levels and peak cTnI levels. Patients who had MCE had higher cTnI level. Thus, peak cTnI had diagnostic and prognostic value for ACS.
Key concepts: Medicine, Troponin I, Cardiology, Internal medicine, Ejection fraction, Coronary artery disease, Acute coronary syndrome, Troponin