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2002•Unpublished venueRequires access

The Protective Effect of APS against Acute Chemical liver Injury of the Mouse

Zheng Zuo

Open publisher page 2 citations

Abstract

To study protective effect of APS against acute chemical liver injury.An animal model with acute liver injury was reproduced by carbon tetachloride and D chondrosamine in mice.Liver function indicators,serum alanine aminotransferase(ALT) and aspartate aminotransferase(AST),were determined.Increase in serum ALTand AST in mice caused by carbon tetrachloride and D chondrosamine could be inhibited by APS. There was significant difference between them and control.[Conclusion]It suggests that APS can protect mice from acute chemical liver injury.

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What this paper is about

To study protective effect of APS against acute chemical liver injury.An animal model with acute liver injury was reproduced by carbon tetachloride and D chondrosamine in mice.Liver function indicators,serum alanine aminotransferase(ALT) and aspartate aminotransferase(AST),were determined.Increase in serum ALTand AST in mice caused by carbon tetrachloride and D chondrosamine could be inhibited by APS. There was significant difference between them and control.[Conclusion]It suggests that APS can protect mice from acute chemical liver injury.

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OpenAlex reports 2 citations for this work. Citation counts describe recorded attention and do not establish research quality.

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Available abstract

To study protective effect of APS against acute chemical liver injury.An animal model with acute liver injury was reproduced by carbon tetachloride and D chondrosamine in mice.Liver function indicators,serum alanine aminotransferase(ALT) and aspartate aminotransferase(AST),were determined.Increase in serum ALTand AST in mice caused by carbon tetrachloride and D chondrosamine could be inhibited by APS. There was significant difference between them and control.[Conclusion]It suggests that APS can protect mice from acute chemical liver injury.

Key concepts: Carbon tetrachloride, Liver injury, Alanine aminotransferase, Medicine, Liver damage, Alanine, Acute exposure, Liver function

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