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2017•PubMedRequires access

[Differences of Therapeutic Efficacy Between Different Kinds of Somatostatin Analogue for Primary Hepatocellular Carcinoma].

Xiu-He Lü, Chunhui Wang, Yan Xie

Open publisher page 2 citations

Abstract

OBJECTIVE: , and to analyze the differences of their therapeutic efficacy with relevant mechanisms. METHODS: in tumor tissues from the level of gene and protein. RESULTS: <0.05). The Western blot test confirmed these results from the protein level. CONCLUSION: In a certain concentration range, the long-term treatment of SSTA with high affinities to SSTR2 and SSTR5 could inhibit the growth of HCC.

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What this paper is about

OBJECTIVE: , and to analyze the differences of their therapeutic efficacy with relevant mechanisms. METHODS: in tumor tissues from the level of gene and protein. RESULTS: <0.05). The Western blot test confirmed these results from the protein level. CONCLUSION: In a certain concentration range, the long-term treatment of SSTA with high affinities to SSTR2 and SSTR5 could inhibit the growth of HCC.

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Available abstract

OBJECTIVE: , and to analyze the differences of their therapeutic efficacy with relevant mechanisms. METHODS: in tumor tissues from the level of gene and protein. RESULTS: <0.05). The Western blot test confirmed these results from the protein level. CONCLUSION: In a certain concentration range, the long-term treatment of SSTA with high affinities to SSTR2 and SSTR5 could inhibit the growth of HCC.

Key concepts: Lanreotide, In vivo, Apoptosis, Somatostatin receptor 2, Somatostatin, Hepatocellular carcinoma, Cell counting, Cell growth

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[Differences of Therapeutic Efficacy Between Different Kinds of Somatostatin Analogue for Primary Hepatocellular Carcinoma]. — Research Paper | ScholarLens