2005Unpublished venueRequires access

Clinical analysis of 163 cases of hemorrhagical cerebral infaction

Liu Xi-can

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Abstract

Objective To explore etiology and clinical features of hemorrhagical cerebral infarction(HCI).Methods CT and clinical features of 136 patients with HCI were analyzed.Results The major etiology included cardiogenic cerebral embolism(39.7%),hypertension(32.4%), hyperglycaemia(25.0%),unknown etiology(2.9%).Among of over 70 years old patients,history of anticoagulation,thrombolysis and hyperglycaemia in patients with haematoma were obviously more than those in patients without haematoma(P0.05). Total effective rate in patients,which bleeding time was shorter than 3d after cerebral infarction,was obviously lower than that of group of bleeding time was 3~7 d(P0.05) and group of bleeding time was over 7 d (P0.01),there was no obvious difference in group of bleeding time was 3~7 d and group that in of bleeding time over 7 d.In 44 patients with haematoma ,24 cases was effective(54.5%),In 92 patients without haematoma,86 cases was effective(93.5%),Effective rate of patients without haematoma was obviously higher than that of patients with haematoma(P0.01).Conclusion Etiology of HCI mostly is cardiogenic cerebral embolism,advanced age, hyperglycaemia,history of anticoagulation and thrombolysis treatment.In patients with more shorter interval from HCI onset to ischemic stroke, the symptom and prognosis is worse.

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Objective To explore etiology and clinical features of hemorrhagical cerebral infarction(HCI).Methods CT and clinical features of 136 patients with HCI were analyzed.Results The major etiology included cardiogenic cerebral embolism(39.7%),hypertension(32.4%), hyperglycaemia(25.0%),unknown etiology(2.9%).Among of over 70 years old patients,history of anticoagulation,thrombolysis and hyperglycaemia in patients with haematoma were obviously more than those in patients without haematoma(P0.05). Total effective rate in patients,which bleeding time was shorter than 3d after cerebral infarction,was obviously lower than that of group of bleeding time was 3~7 d(P0.05) and group of bleeding time was over 7 d (P0.01),there was no obvious difference in group of bleeding time was 3~7 d and group that in of bleeding time over 7 d.In 44 patients with haematoma ,24 cases was effective(54.5%),In 92 patients without haematoma,86 cases was effective(93.5%),Effective rate of patients without haematoma was obviously higher than that of patients with haematoma(P0.01).Conclusion Etiology of HCI mostly is cardiogenic cerebral embolism,advanced age, hyperglycaemia,history of anticoagulation and thrombolysis treatment.In patients with more shorter interval from HCI onset to ischemic stroke, the symptom and prognosis is worse.

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Available abstract

Objective To explore etiology and clinical features of hemorrhagical cerebral infarction(HCI).Methods CT and clinical features of 136 patients with HCI were analyzed.Results The major etiology included cardiogenic cerebral embolism(39.7%),hypertension(32.4%), hyperglycaemia(25.0%),unknown etiology(2.9%).Among of over 70 years old patients,history of anticoagulation,thrombolysis and hyperglycaemia in patients with haematoma were obviously more than those in patients without haematoma(P0.05). Total effective rate in patients,which bleeding time was shorter than 3d after cerebral infarction,was obviously lower than that of group of bleeding time was 3~7 d(P0.05) and group of bleeding time was over 7 d (P0.01),there was no obvious difference in group of bleeding time was 3~7 d and group that in of bleeding time over 7 d.In 44 patients with haematoma ,24 cases was effective(54.5%),In 92 patients without haematoma,86 cases was effective(93.5%),Effective rate of patients without haematoma was obviously higher than that of patients with haematoma(P0.01).Conclusion Etiology of HCI mostly is cardiogenic cerebral embolism,advanced age, hyperglycaemia,history of anticoagulation and thrombolysis treatment.In patients with more shorter interval from HCI onset to ischemic stroke, the symptom and prognosis is worse.

Key concepts: Medicine, Etiology, Thrombolysis, Cerebral infarction, Cerebral embolism, Surgery, Embolism, Internal medicine

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