2004The Chinese Journal of Cardiac Pacing and ElectrophysiologyRequires access

The Clinical and Electrocardiographic Character of Abnormal J Wave and Brugada′s Syndrome or Electrocardiogram Sign of Idiopathic Brugada′s

Xiaoshen Hu

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Abstract

We investigated the clinical and electrocardiographic character of 21 cases with the abnormal J wave,8 cases with the Brugada′s syndrome,and 11 cases with the electrocardiogram sign of idiopathic Brugada′s. Results: ①Except in lead aVR(aVL in a part patients), there were upright abnormal J wave in limb or(and) precordal leads in idiopathic and secondary abnormal J wave. The J waves were significantly lower and distributived extensiver in idiopathic abnormal J wave than those in Brugada′s syndrome and electrocardiogram sign of idiopathic Brugada′s. There was not J waves generally in leads V 1~V 2, if it had , which were smaller than those R waves.The T waves often was upright and without right bundle branch block(RBBB) in leads V 1~V 3. There was explicit etiology in secondary abnormal J wave, their J waves were significantly higher and distributived extensiver and that also had longer QTmin、QTmax、STmax than those in idiopathic abnormal J wave ,the Brugada′s syndrome,and the electrocardiogram sign of idiopathic Brugada′s. In both instances polymorphic ventricular tachycardia and ventricular fibrillation were more often induced and led to death. ②The ST segment was elevation of slanting type or saddle type with RBBB or similar RBBB in the Brugada′s syndrome and the electrocardiogram sign of idiopathic Brugada′s . Their R′ waves was higher than R waves in leads V 1~V 3, T waves often was inversion or upright in leads V 1~V 3.The former appeared usually sudden cardiac death or cardiac syncope by malignant rapid ventricular arrhythmia and the latter had not. Conclusion: The idiopathic and secondary abnormal J wave are different from the Brugada′s syndrome and the electrocardiogram sign of idiopathic Brugada′s in clinical and electrocardiographic character.

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What this paper is about

We investigated the clinical and electrocardiographic character of 21 cases with the abnormal J wave,8 cases with the Brugada′s syndrome,and 11 cases with the electrocardiogram sign of idiopathic Brugada′s. Results: ①Except in lead aVR(aVL in a part patients), there were upright abnormal J wave in limb or(and) precordal leads in idiopathic and secondary abnormal J wave. The J waves were significantly lower and distributived extensiver in idiopathic abnormal J wave than those in Brugada′s syndrome and electrocardiogram sign of idiopathic Brugada′s. There was not J waves generally in leads V 1~V 2, if it had , which were smaller than those R waves.The T waves often was upright and without right bundle branch block(RBBB) in leads V 1~V 3. There was explicit etiology in secondary abnormal J wave, their J waves were significantly higher and distributived extensiver and that also had longer QTmin、QTmax、STmax than those in idiopathic abnormal J wave ,the Brugada′s syndrome,and the electrocardiogram sign of idiopathic Brugada′s. In both instances polymorphic ventricular tachycardia and ventricular fibrillation were more often induced and led to death. ②The ST segment was elevation of slanting type or saddle type with RBBB or similar RBBB in the Brugada′s syndrome and the electrocardiogram sign of idiopathic Brugada′s . Their R′ waves was higher than R waves in leads V 1~V 3, T waves often was inversion or upright in leads V 1~V 3.The former appeared usually sudden cardiac death or cardiac syncope by malignant rapid ventricular arrhythmia and the latter had not. Conclusion: The idiopathic and secondary abnormal J wave are different from the Brugada′s syndrome and the electrocardiogram sign of idiopathic Brugada′s in clinical and electrocardiographic character.

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Available abstract

We investigated the clinical and electrocardiographic character of 21 cases with the abnormal J wave,8 cases with the Brugada′s syndrome,and 11 cases with the electrocardiogram sign of idiopathic Brugada′s. Results: ①Except in lead aVR(aVL in a part patients), there were upright abnormal J wave in limb or(and) precordal leads in idiopathic and secondary abnormal J wave. The J waves were significantly lower and distributived extensiver in idiopathic abnormal J wave than those in Brugada′s syndrome and electrocardiogram sign of idiopathic Brugada′s. There was not J waves generally in leads V 1~V 2, if it had , which were smaller than those R waves.The T waves often was upright and without right bundle branch block(RBBB) in leads V 1~V 3. There was explicit etiology in secondary abnormal J wave, their J waves were significantly higher and distributived extensiver and that also had longer QTmin、QTmax、STmax than those in idiopathic abnormal J wave ,the Brugada′s syndrome,and the electrocardiogram sign of idiopathic Brugada′s. In both instances polymorphic ventricular tachycardia and ventricular fibrillation were more often induced and led to death. ②The ST segment was elevation of slanting type or saddle type with RBBB or similar RBBB in the Brugada′s syndrome and the electrocardiogram sign of idiopathic Brugada′s . Their R′ waves was higher than R waves in leads V 1~V 3, T waves often was inversion or upright in leads V 1~V 3.The former appeared usually sudden cardiac death or cardiac syncope by malignant rapid ventricular arrhythmia and the latter had not. Conclusion: The idiopathic and secondary abnormal J wave are different from the Brugada′s syndrome and the electrocardiogram sign of idiopathic Brugada′s in clinical and electrocardiographic character.

Key concepts: Brugada syndrome, Medicine, J wave, Cardiology, Internal medicine, Right bundle branch block, Electrocardiography, Ventricular fibrillation

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