2012•The International Journal of Biochemistry & Cell BiologyOpen access

Targeted disruption of the p160 coactivator interface of androgen receptor (AR) selectively inhibits AR activity in both androgen-dependent and castration-resistant AR-expressing prostate cancer cells

Manjula Nakka, Irina U. Agoulnik, Nancy L. Weigel

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Key concepts: LNCaP, Androgen receptor, Coactivator, Prostate cancer, TMPRSS2, Cancer research, Androgen, Proto-oncogene tyrosine-protein kinase Src

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Targeted disruption of the p160 coactivator interface of androgen receptor (AR) selectively inhibits AR activity in both androgen-dependent and castration-resistant AR-expressing prostate cancer cells — Research Paper | ScholarLens