2013Journal of Cataract & Refractive SurgeryRequires access

Interface fluid syndrome after laser in situ keratomileusis following herpetic keratouveitis

So Goto, Shizuka Koh, Ryotaro Toda, Takeshi Soma, Kenji Matsushita, Naoyuki Maeda, Kohji Nishida

Open publisher page 13 citations

Abstract

We report an unusual case of a 28-year-old man who developed interface fluid with herpetic keratouveitis and elevated intraocular pressure (IOP) after laser in situ keratomileusis (LASIK) in the left eye. The IOP was unmeasurable with the Goldmann applanation tonometer; rebound tonometry showed an IOP of 30 mm Hg in the central cornea and 58 mm Hg in the superior cornea. Medical treatment of the elevated IOP resulted in resolution of the accumulated interface fluid. This case highlights the inaccuracy of IOP measurements in the central cornea with interface fluid or central corneal edema caused by elevated IOP after LASIK and shows the efficacy of IOP measurements at the peripheral cornea using rebound tonometry. It also shows that uveitis can be a risk factor for interface fluid syndrome after LASIK.

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What this paper is about

We report an unusual case of a 28-year-old man who developed interface fluid with herpetic keratouveitis and elevated intraocular pressure (IOP) after laser in situ keratomileusis (LASIK) in the left eye. The IOP was unmeasurable with the Goldmann applanation tonometer; rebound tonometry showed an IOP of 30 mm Hg in the central cornea and 58 mm Hg in the superior cornea. Medical treatment of the elevated IOP resulted in resolution of the accumulated interface fluid. This case highlights the inaccuracy of IOP measurements in the central cornea with interface fluid or central corneal edema caused by elevated IOP after LASIK and shows the efficacy of IOP measurements at the peripheral cornea using rebound tonometry. It also shows that uveitis can be a risk factor for interface fluid syndrome after LASIK.

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Available abstract

We report an unusual case of a 28-year-old man who developed interface fluid with herpetic keratouveitis and elevated intraocular pressure (IOP) after laser in situ keratomileusis (LASIK) in the left eye. The IOP was unmeasurable with the Goldmann applanation tonometer; rebound tonometry showed an IOP of 30 mm Hg in the central cornea and 58 mm Hg in the superior cornea. Medical treatment of the elevated IOP resulted in resolution of the accumulated interface fluid. This case highlights the inaccuracy of IOP measurements in the central cornea with interface fluid or central corneal edema caused by elevated IOP after LASIK and shows the efficacy of IOP measurements at the peripheral cornea using rebound tonometry. It also shows that uveitis can be a risk factor for interface fluid syndrome after LASIK.

Key concepts: LASIK, Keratomileusis, Cornea, Ophthalmology, Medicine, Intraocular pressure, Applanation tonometry, Optometry

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