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2011•Elsevier eBooksRequires access

Contributions of Genetically Modified Mouse Models to Understanding the Physiology and Pathophysiology of the 25-Hydroxyvitamin D-1-Alpha Hydroxylase Enzyme (1α(OH)ase) and the Vitamin D Receptor (VDR)

Geoffrey N. Hendy, Richard Kremer, David Goltzman

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Key concepts: Calcitriol receptor, Vitamin D and neurology, Receptor, Enzyme, Vitamin, Chemistry, Hydroxylation, Endocrinology

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Contributions of Genetically Modified Mouse Models to Understanding the Physiology and Pathophysiology of the 25-Hydroxyvitamin D-1-Alpha Hydroxylase Enzyme (1α(OH)ase) and the Vitamin D Receptor (VDR) — Research Paper | ScholarLens