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2023•Unpublished venueOpen access

Consideration of serum IL-36α and β levels trends in two patients with chikungunya fever

Makoto Kondo, Yoshiaki Matsushima, Takehisa Nakanishi, Shohei Iida, Koji Habe, Keiichi Yamanaka

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Abstract

1. IntroductionChikungunya fever is a persistent joint pain disease caused by mosquito-borne arboviruses: chikungunya virus (CHIKV). In Chikungunya fever, inflammatory cytokines such as IL-6, Il-17, IL-22, and IL-23 ［1,2］ are involved in chronic joint pain, but there are no reports on whether IL-36 is specified as a cause of joint pain. This case study compares the changes in IL-36α and β levels during infected chikungunya fever and the recovery. This article highlights the relationship between chikungunya fever and IL-36.

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1. IntroductionChikungunya fever is a persistent joint pain disease caused by mosquito-borne arboviruses: chikungunya virus (CHIKV). In Chikungunya fever, inflammatory cytokines such as IL-6, Il-17, IL-22, and IL-23 ［1,2］ are involved in chronic joint pain, but there are no reports on whether IL-36 is specified as a cause of joint pain. This case study compares the changes in IL-36α and β levels during infected chikungunya fever and the recovery. This article highlights the relationship between chikungunya fever and IL-36.

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Available abstract

1. IntroductionChikungunya fever is a persistent joint pain disease caused by mosquito-borne arboviruses: chikungunya virus (CHIKV). In Chikungunya fever, inflammatory cytokines such as IL-6, Il-17, IL-22, and IL-23 ［1,2］ are involved in chronic joint pain, but there are no reports on whether IL-36 is specified as a cause of joint pain. This case study compares the changes in IL-36α and β levels during infected chikungunya fever and the recovery. This article highlights the relationship between chikungunya fever and IL-36.

Key concepts: Chikungunya, Chikungunya fever, Joint pain, Virus, Medicine, Virology, Immunology, Disease

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