Richard Albert, David A. Schwartz
Abstract
Pathogenic findings in a mouse model of idiopathic pulmonary fibrosis confirm the pivotal role played by dysfunction of the type 2 alveolar (AT2) epithelial cells in causing the disease. The findings support the use of this model in future investigations.
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Pathogenic findings in a mouse model of idiopathic pulmonary fibrosis confirm the pivotal role played by dysfunction of the type 2 alveolar (AT2) epithelial cells in causing the disease. The findings support the use of this model in future investigations.
Key concepts: Idiopathic pulmonary fibrosis, Pulmonary fibrosis, Pulmonary disease, Fibrosis, Disease, Medicine, Pathology, Immunology