ScholarLens Back to search
2018•International Journal of CardiologyRequires access

Extracellular signal-regulated kinase (ERK) activation preserves cardiac function in pressure overload induced hypertrophy

Michael Mutlak, Michal Schlesinger−Laufer, Tali Haas, Rona Shofti, Nimer Ballan, Yair E. Lewis, Mor Zuler, Yaniv Zohar, Lilac Caspi, Izhak Kehat

Open publisher page 45 citations

Abstract

This record does not include an abstract. Use the full-text link above if available.

About this research paper

What this paper is about

An abstract is not available in the OpenAlex record for this paper.

Why it matters

OpenAlex reports 45 citations for this work. Citation counts describe recorded attention and do not establish research quality.

Key contribution

A contribution statement is not available in the OpenAlex record.

Method / approach

Method details are not available in the OpenAlex metadata.

Main findings

Findings are not separately available in the OpenAlex metadata.

Limitations

Limitations are not available in the OpenAlex metadata.

Applications

Application details are not available in the OpenAlex metadata.

Key concepts: Pressure overload, Muscle hypertrophy, Medicine, Fibrosis, MAPK/ERK pathway, Internal medicine, Heart failure, Extracellular

Related papers

Related paper 1Related paper 2
Back to paper searchBrowse research topicsOriginal source
Extracellular signal-regulated kinase (ERK) activation preserves cardiac function in pressure overload induced hypertrophy — Research Paper | ScholarLens