Earthquake Source Process and Aftershock Activity
Takashi Miyatake
Abstract
Open-access reader
Takashi Miyatake
Abstract
Open-access reader
In many earthquakes, aftershock activity tends to cluster near the endzone of the fault. This clustering is expected from elastic theory, i. e., two dimensional crack causes high stress region near two endzones of the fault. But, in many shallow strike-slip earthquakes, we can find only one of them. In this paper, we will solve why the clustering is activated near either of the endzones. We study aftershock distributions and source processes of inland long, shallow faults in which we can easily distinguish the clustering near the endzones. It is shown that in bilateral case we can see two clusters at two endpoints of rupture propagation, but in unilateral case one cluster at an endpoint of rupture. The dynamic crack model shows that accelaration, particle velocity and dynamic stress change near the stopping point of rupture are larger than those near the starting point. This may cause much activation of aftershocks near the stopping point of rupture than near the starting point, i. e., hypocenter of main shock.
OpenAlex reports 6 citations for this work. Citation counts describe recorded attention and do not establish research quality.
A contribution statement is not available in the OpenAlex record.
Method details are not available in the OpenAlex metadata.
Findings are not separately available in the OpenAlex metadata.
Limitations are not available in the OpenAlex metadata.
Application details are not available in the OpenAlex metadata.
In many earthquakes, aftershock activity tends to cluster near the endzone of the fault. This clustering is expected from elastic theory, i. e., two dimensional crack causes high stress region near two endzones of the fault. But, in many shallow strike-slip earthquakes, we can find only one of them. In this paper, we will solve why the clustering is activated near either of the endzones. We study aftershock distributions and source processes of inland long, shallow faults in which we can easily distinguish the clustering near the endzones. It is shown that in bilateral case we can see two clusters at two endpoints of rupture propagation, but in unilateral case one cluster at an endpoint of rupture. The dynamic crack model shows that accelaration, particle velocity and dynamic stress change near the stopping point of rupture are larger than those near the starting point. This may cause much activation of aftershocks near the stopping point of rupture than near the starting point, i. e., hypocenter of main shock.
Key concepts: Aftershock, Seismology, Hypocenter, Geology, Foreshock, Cluster analysis, Shock (circulatory), Slip (aerodynamics)