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2012•Progress in Veterinary MedicineRequires access

Progress on Cytochrome C and Cell Apoptosis

Pengfei Zhang

Open publisher page 1 citations

Abstract

In the process of apoptosis,mitochondria are the apoptosis regulation center,and cytochrome C(Cyt-C) plays a key role in the release from mitochondria.Cytochrome C can activate caspase when cytochrome C is released into the cytoplasm,leading to apoptosis.The Bcl-2 protein family can regulates cytochrome C release.Apoptosis inducing factor AIF ensures the apoptosis in an orderly manner.Endoplasmic reticulum can improve the mitochondrial pro-apoptotic factor sensitivity through stress,recruitment,activation,therefore plays an important role in the release of cytochrome C from mitochondrial IMS.

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What this paper is about

In the process of apoptosis,mitochondria are the apoptosis regulation center,and cytochrome C(Cyt-C) plays a key role in the release from mitochondria.Cytochrome C can activate caspase when cytochrome C is released into the cytoplasm,leading to apoptosis.The Bcl-2 protein family can regulates cytochrome C release.Apoptosis inducing factor AIF ensures the apoptosis in an orderly manner.Endoplasmic reticulum can improve the mitochondrial pro-apoptotic factor sensitivity through stress,recruitment,activation,therefore plays an important role in the release of cytochrome C from mitochondrial IMS.

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Available abstract

In the process of apoptosis,mitochondria are the apoptosis regulation center,and cytochrome C(Cyt-C) plays a key role in the release from mitochondria.Cytochrome C can activate caspase when cytochrome C is released into the cytoplasm,leading to apoptosis.The Bcl-2 protein family can regulates cytochrome C release.Apoptosis inducing factor AIF ensures the apoptosis in an orderly manner.Endoplasmic reticulum can improve the mitochondrial pro-apoptotic factor sensitivity through stress,recruitment,activation,therefore plays an important role in the release of cytochrome C from mitochondrial IMS.

Key concepts: Cytochrome c, Apoptosis, Mitochondrion, Cell biology, Apoptosome, Cytochrome, Mitochondrial apoptosis-induced channel, Apoptosis-inducing factor

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