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Research Advancement of Pathogenesis of Hepatits B Virus-associated Glomerulonephritis

Wenhu Liu

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Abstract

The pathogenesis of hepatitis B virus-associated glomerulonephritis(HBV-GN) is not precisely defined.Five major mechanisms could induce renal tissue injury by hepatitis B virus(HBV):(1) Tissue deposition of immune complex of viral antigen and host antibody was accepted widely.(2) Cytopathic effect induced by virus infection of the cell.(3) Immunodeficiency induced by HBV infection could result in HBV persisted in the host.(4) HBV mutation could enhance the invasion ability of virus.(5) Genetic factors have been proposed as playing a role in the pathogenesis of HBV-GN.

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What this paper is about

The pathogenesis of hepatitis B virus-associated glomerulonephritis(HBV-GN) is not precisely defined.Five major mechanisms could induce renal tissue injury by hepatitis B virus(HBV):(1) Tissue deposition of immune complex of viral antigen and host antibody was accepted widely.(2) Cytopathic effect induced by virus infection of the cell.(3) Immunodeficiency induced by HBV infection could result in HBV persisted in the host.(4) HBV mutation could enhance the invasion ability of virus.(5) Genetic factors have been proposed as playing a role in the pathogenesis of HBV-GN.

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Available abstract

The pathogenesis of hepatitis B virus-associated glomerulonephritis(HBV-GN) is not precisely defined.Five major mechanisms could induce renal tissue injury by hepatitis B virus(HBV):(1) Tissue deposition of immune complex of viral antigen and host antibody was accepted widely.(2) Cytopathic effect induced by virus infection of the cell.(3) Immunodeficiency induced by HBV infection could result in HBV persisted in the host.(4) HBV mutation could enhance the invasion ability of virus.(5) Genetic factors have been proposed as playing a role in the pathogenesis of HBV-GN.

Key concepts: Pathogenesis, Hepatitis B virus, Virology, Virus, Glomerulonephritis, Hepatitis B, Immunology, Antibody

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