Ipsilateral Hemiparesis Caused by a Internal Capsule Infarct after a Previous Stroke on the Opposite Side
Tae Hak Kim, Cheol Seung Shin, Kee Ra Lee, Seung‐Han Lee, Seong‐Min Choi, Man‐Seok Park, Byeong Chae Kim, Myeong Kyu Kim, Ki Hyun Cho
Abstract
Tae Hak Kim, Cheol Seung Shin, Kee Ra Lee, Seung‐Han Lee, Seong‐Min Choi, Man‐Seok Park, Byeong Chae Kim, Myeong Kyu Kim, Ki Hyun Cho
Abstract
A 63-year-old man was admitted with newly developed left hemiparesis. He had experienced left hemiparesis that had resulted from an earlier right thalamic hemorrhage. Diffusion-weighted images showed only high signal intensity lesion in the left internal capsule. Ipsilateral motor evoked potentials were obtained at the affected hand muscles when the unaffected motor cortex was stimulated. This suggests that cortical reorganization in the unaffected hemisphere after the first stroke may be involved in the occurrence of such symptom. J Korean Neurol Assoc 24(5):468-471, 2006
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A 63-year-old man was admitted with newly developed left hemiparesis. He had experienced left hemiparesis that had resulted from an earlier right thalamic hemorrhage. Diffusion-weighted images showed only high signal intensity lesion in the left internal capsule. Ipsilateral motor evoked potentials were obtained at the affected hand muscles when the unaffected motor cortex was stimulated. This suggests that cortical reorganization in the unaffected hemisphere after the first stroke may be involved in the occurrence of such symptom. J Korean Neurol Assoc 24(5):468-471, 2006
Key concepts: Internal capsule, Hemiparesis, Medicine, Lesion, Stroke (engine), Motor cortex, Lateralization of brain function, Cardiology