2009Thrombosis JournalOpen access

Hemostasis and inflammation: two of a kind?

Peter Verhamme, Marc Hoylaerts

Open full text 69 citations

Abstract

Hemostasis is a defense mechanism to stop bleeding. Activated by vessel wall injury, it consists of intertwined activation of platelets and the coagulation cascade, tightly controlled by natural anticoagulants and the fibrinolytic system.

Open-access reader

About this research paper

What this paper is about

Hemostasis is a defense mechanism to stop bleeding. Activated by vessel wall injury, it consists of intertwined activation of platelets and the coagulation cascade, tightly controlled by natural anticoagulants and the fibrinolytic system.

Why it matters

OpenAlex reports 69 citations for this work. Citation counts describe recorded attention and do not establish research quality.

Key contribution

A contribution statement is not available in the OpenAlex record.

Method / approach

Method details are not available in the OpenAlex metadata.

Main findings

Findings are not separately available in the OpenAlex metadata.

Limitations

Limitations are not available in the OpenAlex metadata.

Applications

Application details are not available in the OpenAlex metadata.

Available abstract

Hemostasis is a defense mechanism to stop bleeding. Activated by vessel wall injury, it consists of intertwined activation of platelets and the coagulation cascade, tightly controlled by natural anticoagulants and the fibrinolytic system.

Key concepts: Hemostasis, Medicine, Coagulation cascade, Inflammation, Angiology, Coagulation, Hematology, Platelet

Related papers

Back to paper searchBrowse research topicsOriginal source
Hemostasis and inflammation: two of a kind? — Research Paper | ScholarLens