Vincent Hascall, Aimin Wang
Abstract
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This commentary proposes a mechanism for why murine diabetic adipose tissue contains very few remaining stem cells compared with normal adipose tissue. The mechanism involves the diversion of stem cells to pathological adipocytes when they divide in hyperglycemia.
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This commentary proposes a mechanism for why murine diabetic adipose tissue contains very few remaining stem cells compared with normal adipose tissue. The mechanism involves the diversion of stem cells to pathological adipocytes when they divide in hyperglycemia.
Key concepts: Adipose tissue, Stem cell, Adipogenesis, Pathological, Mechanism (biology), Cell biology, Biology, Medicine