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2004•Neurobiology of AgingRequires access

P4-312 The Ca2+ overload hypothesis in the familial form of Alzheimer's disease: new light from presenilin 2 mutations

Cristina Fasolato, Giancarlo Zatti, Marta Giacomello, Roberta Ghidoni, Laura Barbiero, Giuliano Binetti, Paola Pizzo

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Key concepts: Thapsigargin, SERCA, Cyclopiazonic acid, Ryanodine receptor, Chemistry, Endoplasmic reticulum, Ruthenium red, Ionomycin

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P4-312 The Ca2+ overload hypothesis in the familial form of Alzheimer's disease: new light from presenilin 2 mutations — Research Paper | ScholarLens