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1970•The Journal of Clinical Endocrinology & MetabolismRequires access

Suppressibility of Thyroid Function During Treatment with Antithyroid Drugs in Graves' Disease

J. Wils, P. W. C. Kloppenborg

Open publisher page 2 citations

Abstract

A high radioiodine uptake in the presence of a normal PBI, 24 hr after discontinuing antithyroid drug treatment in patients with Graves' disease, should not necessarily be interpreted to be a sign of gland autonomy. It is shown that identical findings can occur in normal subjects incorrectly treated with antithyroid drugs. The combination of these findings might be explained easily as the consequence of TSH overstimulation.

About this research paper

What this paper is about

A high radioiodine uptake in the presence of a normal PBI, 24 hr after discontinuing antithyroid drug treatment in patients with Graves' disease, should not necessarily be interpreted to be a sign of gland autonomy. It is shown that identical findings can occur in normal subjects incorrectly treated with antithyroid drugs. The combination of these findings might be explained easily as the consequence of TSH overstimulation.

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OpenAlex reports 2 citations for this work. Citation counts describe recorded attention and do not establish research quality.

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Available abstract

A high radioiodine uptake in the presence of a normal PBI, 24 hr after discontinuing antithyroid drug treatment in patients with Graves' disease, should not necessarily be interpreted to be a sign of gland autonomy. It is shown that identical findings can occur in normal subjects incorrectly treated with antithyroid drugs. The combination of these findings might be explained easily as the consequence of TSH overstimulation.

Key concepts: Antithyroid drugs, Medicine, Graves' disease, Internal medicine, Antithyroid agent, Endocrinology, Thyroid function, Thyroid

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Suppressibility of Thyroid Function During Treatment with Antithyroid Drugs in Graves' Disease — Research Paper | ScholarLens