2003Cancer Genetics and CytogeneticsOpen access

Different mechanisms lead to a karyotypically identical t(20;21) in myelodysplastic syndrome and in acute myelocytic leukemia

Caterina Matteucci, Roberta La Starza, Barbara Crescenzi, Silvia Romoli, Alessandra Santoro, Silvana Magrin, Francesco Lauria, Francesco Lo‐Coco, Massimo F. Martelli, Cristina Mecucci

Open full text 7 citations

Abstract

This record does not include an abstract. Use the full-text link above if available.

Open-access reader

About this research paper

What this paper is about

An abstract is not available in the OpenAlex record for this paper.

Why it matters

OpenAlex reports 7 citations for this work. Citation counts describe recorded attention and do not establish research quality.

Key contribution

A contribution statement is not available in the OpenAlex record.

Method / approach

Method details are not available in the OpenAlex metadata.

Main findings

Findings are not separately available in the OpenAlex metadata.

Limitations

Limitations are not available in the OpenAlex metadata.

Applications

Application details are not available in the OpenAlex metadata.

Key concepts: Biology, Fluorescence in situ hybridization, Subtelomere, Chromosomal translocation, Myelocytic leukemia, Comparative genomic hybridization, Locus (genetics), Karyotype

Related papers

Back to paper searchBrowse research topicsOriginal source
Different mechanisms lead to a karyotypically identical t(20;21) in myelodysplastic syndrome and in acute myelocytic leukemia — Research Paper | ScholarLens