2007•Biochemical and Biophysical Research CommunicationsRequires access

Rosiglitazone attenuates NF-κB-dependent ICAM-1 and TNF-α production caused by homocysteine via inhibiting ERK1/2/p38MAPK activation

Yongping Bai, Yuhui Liu, Jia Chen, Tao Song, Yu You, Zhen-Yan Tang, Yuan‐Jian Li, Guogang Zhang

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Key concepts: MAPK/ERK pathway, Umbilical vein, NF-κB, Staurosporine, Tumor necrosis factor alpha, Chemistry, Signal transduction, Rosiglitazone

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Rosiglitazone attenuates NF-κB-dependent ICAM-1 and TNF-α production caused by homocysteine via inhibiting ERK1/2/p38MAPK activation — Research Paper | ScholarLens