2012•Biochimica et Biophysica Acta (BBA) - Molecular Basis of DiseaseOpen access

Metabolically induced heteroplasmy shifting and l-arginine treatment reduce the energetic defect in a neuronal-like model of MELAS

Valérie Desquiret‐Dumas, Naïg Guéguen, Magalie Barth, Arnaud Chevrollier, Saege A. Hancock, Douglas C. Wallace, Patrizia Amati‐Bonneau, Daniel Henrion, Dominique Bonneau, Pascal Reynier, Vincent Procaccio

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Key concepts: Heteroplasmy, Mitochondrial myopathy, MELAS syndrome, Mitochondrial DNA, Respiratory chain, Lactic acidosis, Biology, Mitochondrial encephalomyopathy