2012•Journal of NeuroimmunologyRequires access

Divalent and monovalent autoantibodies cause dysfunction of MuSK by distinct mechanisms in a rabbit model of myasthenia gravis

Shuuichi Mori, Shigeru Yamada, Sachiho Kubo, Jie Chen, Seiji Matsuda, Masachika Shudou, Naoki Maruyama, Kazuhiro Shigemoto

Open publisher page 45 citations

Abstract

This record does not include an abstract. Use the full-text link above if available.

About this research paper

What this paper is about

An abstract is not available in the OpenAlex record for this paper.

Why it matters

OpenAlex reports 45 citations for this work. Citation counts describe recorded attention and do not establish research quality.

Key contribution

A contribution statement is not available in the OpenAlex record.

Method / approach

Method details are not available in the OpenAlex metadata.

Main findings

Findings are not separately available in the OpenAlex metadata.

Limitations

Limitations are not available in the OpenAlex metadata.

Applications

Application details are not available in the OpenAlex metadata.

Key concepts: Agrin, Myasthenia gravis, Acetylcholine receptor, Antibody, Neuromuscular junction, Autoantibody, Divalent, In vitro

Related papers

Back to paper searchBrowse research topicsOriginal source
Divalent and monovalent autoantibodies cause dysfunction of MuSK by distinct mechanisms in a rabbit model of myasthenia gravis — Research Paper | ScholarLens