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2014•OncogeneRequires access

ATR inhibition preferentially targets homologous recombination-deficient tumor cells

Małgorzata Krajewska, Rudolf S.N. Fehrmann, Pepijn M. Schoonen, Sarah Labib, Elisabeth G.E. de Vries, Lude Franke, Marcel A.T.M. van Vugt

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Key concepts: RAD51, Genome instability, CHEK1, Biology, Homologous recombination, DNA repair, G2-M DNA damage checkpoint, DNA damage

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ATR inhibition preferentially targets homologous recombination-deficient tumor cells — Research Paper | ScholarLens